By Mohammed S. Razzaque, Takashi Taguchi
ISBN-10: 1423796659
ISBN-13: 9781423796657
ISBN-10: 380557858X
ISBN-13: 9783805578585
Read or Download Cellular Stress Responses In Renal Diseases (Contributions to Nephrology) PDF
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Extra resources for Cellular Stress Responses In Renal Diseases (Contributions to Nephrology)
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Kidney Int 1996;50:1202–1211. Uchida S, Nakanishi T, Kwon HM, Preston AS, Handler JS: Taurine behaves as an osmolyte in Madin-Darby canine kidney cells. Protection by polarized, regulated transport of taurine. J Clin Invest 1991;88:656–662. Chen JG, Coe M, McAteer JA, Kempson SA: Hypertonic activation and recovery of system A amino acid transport in renal MDCK cells. Am J Physiol 1996;270:F419–F424. Kempson SA: Differential activation of system A and betaine/GABA transport in MDCK cell membranes by hypertonic stress.
Upregulation of BGT1 in response to hypertonic stress is due mostly to enhanced transcription of the BGT1 gene leading to increased abundance of BGT1 mRNA in the renal medulla [32–35]. Apart from transcriptional regulation of BGT1 expression, cytoskeletal elements (microfilament network and microtubules) may play a role in the control of betaine uptake by modulating BGT1 insertion into the cell membrane [36, 37]. Sorbitol. Enhanced conversion of glucose to sorbitol by aldose reductase (AR) is the principal process leading to the intracellular accumulation of sorbitol in response to hypertonic stress [38].
However, with the implementation of ‘ome’-based technologies, an increasing number of tumor-associated antigens and tumor markers has been identified that includes various heat shock proteins (HSPs). RCC lesions demonstrate heterogeneous expression patterns for HSPs. In most cases overexpression of certain HSPs, such as HSP27, HSP70 and HSP72, has been detected both in RCC cell lines as well as in the tumor lesions when compared to normal kidney epithelium. Furthermore, HSPs play an important role in apoptotic cell death, in the regulation of cell proliferation and in the augmentation of lysis of RCC by HLA class I-restricted cytotoxic T lymphocytes.
Cellular Stress Responses In Renal Diseases (Contributions to Nephrology) by Mohammed S. Razzaque, Takashi Taguchi
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